https://www.genecards.org/cgi-bin/carddisp.pl?gene=AREG
AREG (Amphiregulin) is a Protein Coding gene. Diseases associated with AREG include Ovarian Cystadenoma and Psoriasis.Among its related pathways are Apoptotic Pathways in Synovial Fibroblasts and GPCR Pathway.Gene Ontology (GO) annotations related to this gene include cytokine activity and epidermal growth factor receptor binding.An important paralog of this gene is HBEGF.
https://en.wikipedia.org/wiki/Amphiregulin
Amphiregulin, also known as AREG, is a protein synthesized as a transmembrane glycoprotein with 252 aminoacids and it is encoded by the AREG gene. in humans. Function. The protein encoded by this gene is a member of the epidermal growth factor (EGF) family. It is a
https://pubmed.ncbi.nlm.nih.gov/24463227/
Amphiregulin (AREG) is a ligand of the epidermal growth factor receptor (EGFR), a widely expressed transmembrane tyrosine kinase. AREG is synthesized as a membrane-anchored precursor protein that can engage in juxtacrine signaling on adjacent cells. Alternatively, after proteolytic processing by cel
https://www.aberdeenrenewables.com/
AREG is a not-for-profit organisation that helps its 270+ members access opportunities in renewable energy and champion the sector. Learn more about AREG's vision, services, events, and sponsorship opportunities.
https://pubmed.ncbi.nlm.nih.gov/34698381/
Amphiregulin (AREG), which acts as one of the ligands for epidermal receptor growth factor receptor (EGFR), plays a crucial role in tissue repair, inflammation, and immunity. AREG is synthesized as membrane-anchored pre-protein, and is excreted after proteolytic cleavage, and serves as an autocrine
https://www.sciencedirect.com/science/article/pii/S1074761315000461
AREG has been shown to be of critical importance for efficient CD4 + regulatory T (Treg) cell function, and in the absence of AREG, CD4 + Treg cells cannot suppress local inflammation. As CD4 + Treg cells become activated, they upregulate the EGFR, in part via STAT5-mediated signaling. These activated regulatory T cells migrate to the site of
https://www.nature.com/articles/onc201593
AREG is a low affinity EGFR ligand, which is upregulated following treatment with chemotherapeutic drugs. Because AREG depletion retarded growth of xenografted ovarian tumors in mice, we generated
https://www.ncbi.nlm.nih.gov/gene/374
AREG gene expressed by purified primary myeloma cells, and expression higher than in normal bone marrow (BM) plasma cells or plasmablastic cells; AREG plays an important role in biology of multiple myeloma (TARP) expression in prostate cancer cells leads to an increased growth rate and induction of caveolins and amphiregulin.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10691888/
Amphiregulin (AREG) is a ligand of the epidermal growth factor receptor (EGFR) and has been shown to regulate the phagocytosis-induced cell death of monocytes in peripheral blood. AREG-dependent apoptotic signaling engages factors of the intrinsic and extrinsic apoptotic pathway, such as BCL-2, BCL-XL, and death ligand/receptor CD95/CD95L.
https://www.ncbi.nlm.nih.gov/gene?Cmd=DetailsSearch&Term=374
AREG upregulates secreted protein acidic and rich in cysteine expression in human granulosa cells. Dang X, et al. Mol Cell Endocrinol, 2023 Feb 5. PMID 36462647; Nuclear AREG affects a low-proliferative phenotype and contributes to drug resistance of melanoma. Seefried F, et al. Int J Cancer, 2022 Dec 15. PMID 36054710
https://www.nature.com/articles/s41388-021-01784-8
AREG is highly abundant in abdominal fluids of patients with advanced OvCa. In immunocompetent animals, depletion or overexpression of AREG respectively prolonged or shortened animal survival. A
https://www.areg.org.au/
Posted on 2024-06-02 by VK5QI. One of the services that the Amateur Radio Experimenters Group provides is a network of repeater stations that cover the Adelaide metropolitan and surrounding areas. These systems operate on the 6m-23cm bands from 3 sites in and around Adelaide. One of the first repeaters conceived by AREG was the Adelaide CBD
https://www.sciencedirect.com/science/article/pii/S1084952114000068
Amphiregulin (AREG) is a ligand of the epidermal growth factor receptor (EGFR), a widely expressed transmembrane tyrosine kinase. AREG is synthesized as a membrane-anchored precursor protein that can engage in juxtacrine signaling on adjacent cells. Alternatively, after proteolytic processing by cell membrane proteases, mainly TACE/ADAM17, AREG
https://www.nature.com/articles/onc2015269
These QRT-PCR data also confirm the lack of normalization for these genes by exogenous EGF. Figure 3. Exogenous EGF does not restore expression of FOXM1 and its target genes in response to AREG
https://pubmed.ncbi.nlm.nih.gov/32323797/
Amphiregulin (AREG) is a member of the epidermal growth factor (EGF) family and is expressed in a plethora of cancers. The biological roles of AREG in the regulation of the epithelial‑mesenchymal transition (EMT) in pancreatic cancer remain unclear. To investigate the expression of epidermal growth
https://economics.stackexchange.com/questions/45417/what-are-the-main-differences-among-xtreg-areg-reghdfe
areg circumvents the matsize problem by absorbing the dummies, there is no other significant difference between areg and reg. reghdfe. reghdfe runs linear and instrumental-variable regressions with many levels of fixed effects, by implementing the estimator of Correia (2015) according to the authors of this user written command see here.
https://hiset.org/
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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6826133/
AREG is a novel biomarker imaging in vivo SASP and implies potential immunosuppression in a treatment‐damaged TME. We next sought to assess whether AREG is experimentally detectable in the circulating system of cancer patients post‐treatment. To this end, we collected peripheral blood from PCa patients, including one cohort that experienced
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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4792035/
AREG has been shown to be of critical importance for efficient CD4 + regulatory T (Treg) cell function, and in the absence of AREG, CD4 + Treg cells cannot suppress local inflammation. As CD4 + Treg cells become activated they up-regulate the EGFR, in part via STAT5 mediated signaling. These activated regulatory T cells migrate to the site of
https://onlinelibrary.wiley.com/doi/full/10.1111/acel.13027
AREG is a novel circulating biomarker of in vivo SASP, implies immunosuppression, and predicts adverse therapeutic outcome. (a) Abundance of AREG protein in the serum of untreated and chemo-treated PCa patients. Data were derived from ELISA measurement and shown as mean ± SD, n = 20. (b) Abundance of IL-8 protein in patient serum analyzed in (a).
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